In the battle against intestinal disease, our bodies employ multiple lines of defense to keep harmful bacteria at bay. While most of us never think about these protective mechanisms, people with Crohn’s disease experience what happens when these defenses fail.
A big topic that remains not well-addressed is how norovirus infection triggers disease onset in patients with Crohn’s.
A group of international scientists have successfully identified variants in 10 genes that heighten risk of developing Crohn’s disease.
The results showed the pregnant women with IBD had higher incidences of common pregnancy complications.
Since the 1990s, Leo Otterbein, a professor of surgery at Harvard Medical School and Beth Israel Deaconess Medical Center has been studying the effects of low-dose carbon monoxide.
A group of international researchers say that an antibody detectable in blood can predict severe Crohn’s disease up to seven years prior to diagnosis.
Children who grow up with pet dogs are less likely to get Crohn’s disease later in life, according to a new study.
A new study reveals that the more antibiotics prescribed to patients 60 and older, the more likely they were to develop inflammatory bowel disease (IBD).
The cause of Crohn’s disease is not known. It can occur at any part of the GI tract, from the mouth to the anus.
Neutrophils, a type of white blood cell, fight infection, clear away cell debris, and heal injured tissue. In diseases with chronic or excessive inflammation (e.g., arthritis, sepsis), however, they can also cause tissue damage. Scientists at the University of Illinois, Chicago, recently identified two distinct subtypes of neutrophils, and designed a way to preserve the neutrophils’ favorable effects while their anti-inflammatory properties fight disease.